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α dendrotoxin alomone labs  (Alomone Labs)


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    Alomone Labs α dendrotoxin alomone labs
    α Dendrotoxin Alomone Labs, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 77 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/α dendrotoxin alomone labs/product/Alomone Labs
    Average 94 stars, based on 77 article reviews
    α dendrotoxin alomone labs - by Bioz Stars, 2026-02
    94/100 stars

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    RIPK2-overexpressing PC-3 cells were treated with the NF-κB inhibitor SN50 or P-gp inhibitor tariquidar, and cell sensitivity to <t>DTX</t> (12 nM) was assessed using crystal violet staining ( A ) and flow cytometry (B) . PC-3/DTX cells were treated with SN50, tariquidar, or the RIPK2 <t>inhibitor</t> <t>GSK583,</t> and cell sensitivity to DTX (12 nM) was detected using crystal violet staining ( C ) and flow cytometry (D) . ** P < 0.01. n = 3 in A-D.
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    RIPK2-overexpressing PC-3 cells were treated with the NF-κB inhibitor SN50 or P-gp inhibitor tariquidar, and cell sensitivity to <t>DTX</t> (12 nM) was assessed using crystal violet staining ( A ) and flow cytometry (B) . PC-3/DTX cells were treated with SN50, tariquidar, or the RIPK2 <t>inhibitor</t> <t>GSK583,</t> and cell sensitivity to DTX (12 nM) was detected using crystal violet staining ( C ) and flow cytometry (D) . ** P < 0.01. n = 3 in A-D.
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    RIPK2-overexpressing PC-3 cells were treated with the NF-κB inhibitor SN50 or P-gp inhibitor tariquidar, and cell sensitivity to <t>DTX</t> (12 nM) was assessed using crystal violet staining ( A ) and flow cytometry (B) . PC-3/DTX cells were treated with SN50, tariquidar, or the RIPK2 <t>inhibitor</t> <t>GSK583,</t> and cell sensitivity to DTX (12 nM) was detected using crystal violet staining ( C ) and flow cytometry (D) . ** P < 0.01. n = 3 in A-D.
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    RIPK2-overexpressing PC-3 cells were treated with the NF-κB inhibitor SN50 or P-gp inhibitor tariquidar, and cell sensitivity to <t>DTX</t> (12 nM) was assessed using crystal violet staining ( A ) and flow cytometry (B) . PC-3/DTX cells were treated with SN50, tariquidar, or the RIPK2 <t>inhibitor</t> <t>GSK583,</t> and cell sensitivity to DTX (12 nM) was detected using crystal violet staining ( C ) and flow cytometry (D) . ** P < 0.01. n = 3 in A-D.
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    Image Search Results


    RIPK2-overexpressing PC-3 cells were treated with the NF-κB inhibitor SN50 or P-gp inhibitor tariquidar, and cell sensitivity to DTX (12 nM) was assessed using crystal violet staining ( A ) and flow cytometry (B) . PC-3/DTX cells were treated with SN50, tariquidar, or the RIPK2 inhibitor GSK583, and cell sensitivity to DTX (12 nM) was detected using crystal violet staining ( C ) and flow cytometry (D) . ** P < 0.01. n = 3 in A-D.

    Journal: PLOS One

    Article Title: RIPK2 induces docetaxel resistance in prostate cancer through the NF-κB/P-gp signaling pathway

    doi: 10.1371/journal.pone.0341445

    Figure Lengend Snippet: RIPK2-overexpressing PC-3 cells were treated with the NF-κB inhibitor SN50 or P-gp inhibitor tariquidar, and cell sensitivity to DTX (12 nM) was assessed using crystal violet staining ( A ) and flow cytometry (B) . PC-3/DTX cells were treated with SN50, tariquidar, or the RIPK2 inhibitor GSK583, and cell sensitivity to DTX (12 nM) was detected using crystal violet staining ( C ) and flow cytometry (D) . ** P < 0.01. n = 3 in A-D.

    Article Snippet: When the xenograft tumors grew to approximately 200 mm 3 , the mice were randomly divided into four groups (4 animals per group): DTX (MedChem Express, Monmouth Junction, NJ, USA), DTX + GSK583 (MedChem Express), DTX+tariquidar (MedChem Express), and Control.

    Techniques: Staining, Flow Cytometry

    (A) P-gp and Ki67 expression in xenograft tumor tissues of the control, DTX, DTX + GSK583, and DTX+tariquidar groups. ** P < 0.01. n = 3 in A. (B) Photographs of xenograft tumors in the control, DTX, DTX + GSK583, and DTX+tariquidar groups after 28 days of treatment. (C) Comparison of tumor volumes by group. (D) Comparison of tumor weights by group. ** P < 0.01. n = 4 in B-D.

    Journal: PLOS One

    Article Title: RIPK2 induces docetaxel resistance in prostate cancer through the NF-κB/P-gp signaling pathway

    doi: 10.1371/journal.pone.0341445

    Figure Lengend Snippet: (A) P-gp and Ki67 expression in xenograft tumor tissues of the control, DTX, DTX + GSK583, and DTX+tariquidar groups. ** P < 0.01. n = 3 in A. (B) Photographs of xenograft tumors in the control, DTX, DTX + GSK583, and DTX+tariquidar groups after 28 days of treatment. (C) Comparison of tumor volumes by group. (D) Comparison of tumor weights by group. ** P < 0.01. n = 4 in B-D.

    Article Snippet: When the xenograft tumors grew to approximately 200 mm 3 , the mice were randomly divided into four groups (4 animals per group): DTX (MedChem Express, Monmouth Junction, NJ, USA), DTX + GSK583 (MedChem Express), DTX+tariquidar (MedChem Express), and Control.

    Techniques: Expressing, Control, Comparison